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. 2022 Apr 29;9612033221098571.
doi: 10.1177/09612033221098571.
Online ahead of print.
Affiliations
Affiliations
- 1 1466Johns Hopkins University, Baltimore, MD, USA.
- 2 Johns Hopkins University School of Medicine, Baltimore, MD, USA.
- 3 Hopkins, Baltimore, MD, USA.
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Brigitte Kazzi et al.
Lupus.
.
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. 2022 Apr 29;9612033221098571.
doi: 10.1177/09612033221098571.
Online ahead of print.
Affiliations
- 1 1466Johns Hopkins University, Baltimore, MD, USA.
- 2 Johns Hopkins University School of Medicine, Baltimore, MD, USA.
- 3 Hopkins, Baltimore, MD, USA.
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Abstract
A dysregulated immune response plays a critical role in systemic lupus erythematosus (SLE) pathogenesis. Environmental factors such as viruses, including coronavirus 2 (COVID-19), have been described to play a role in SLE presentation and exacerbation. These viruses trigger a host’s humoral and cellular immunities typically essential in elimination of the viral infection. We present a case of a Hispanic male who developed new-onset lupus nephritis class II after a COVID-19 infection.
Keywords:
COVID-19; lupus nephritis; systemic lupus erythematosus.
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